intermittent heat exposure raises HSP70 and slows arterial clotting with less cardiovascular strain
Progressive thermal preconditioning (three 5-min immersions at 42°C) produced a similar increase in core body temperature and equivalent upregulation of femoral arterial HSP70 expression and delay in FeCl3-induced arterial occlusion as continuous 15-min 42°C immersion, but with significantly less hypertension and tachycardia.
What this means for you
Heat shock protein 70 (HSP70) is one of your body's key repair and protection proteins — when it rises, cells become more resilient to stress and damage. This finding suggests that shorter, repeated heat exposures can trigger the same protective response as longer continuous heat, while putting less demand on your heart and blood pressure — a meaningful distinction for everyday wellness use.
The published research
This page describes a documented finding about the underlying wellness technology. It is not a marketing claim about any specific device. For information about HOCATT specifically, visit hocatt.com.